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Scientists have identified a new cause of hypertension and the path to its treatment through a new study that has provided evidence that a specific region of the brain may be responsible for certain cases of high blood pressure.<\/p>\n
More importantly, researchers believe they have also found a way to reverse this condition. According to this research, carried out by a joint team of the University of S\u00e3o Paulo in Brazil and the University of Auckland in New Zealand, the lateral parafacial region (pFL) of the brain can provoke biological changes that increase blood pressure.<\/p>\n
This region is traditionally known for controlling breathing \u2013 specifically for the forceful and deliberate exhalations of air that occur during exercise, coughing or laughing \u2013 but through laboratory tests, researchers found that it it also has the function of constricting blood vessels.<\/p>\n
This interaction between the control of breathing and the blood vessels may be the main cause of hypertension in many cases, which explains why about 40 percent of people with hypertension fail to keep their blood pressure under control despite existing medications.<\/p>\n
The study suggests that pFL neurons link changes in breathing rhythms to increased activity in the sympathetic nervous system, which controls our “fight” response. or ik,” fitting with previous research linking hypertension to the brain.<\/p>\n
Since about 50 percent of patients with hypertension have a neurogenic component, the challenge was to understand the mechanisms that generate this sympathetic arousal.<\/p>\n
Researchers used genetic engineering techniques in mice to monitor neural activity and found that in hypertensive mice, the pFL region remains constantly active, but when this region was deactivated, blood pressure fell to normal levels.<\/p>\n
These findings also explain why people with sleep apnea have a higher risk of hypertension, as pFL neurons are activated in response to high levels of CO2 or low oxygen.<\/p>\n
Although the research was conducted in animal models, the need for new treatments in humans is urgent to avoid risks such as dementia or heart disease. The next challenge is to target these neurons through the carotid bodies in the neck, using a new drug that can stop their activity remotely without having to penetrate directly into the brain, a strategy that promises greater safety but still requires extensive clinical testing. \/tesheshi.com\/<\/p>\n
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